Covid May Damage Brain Function Major Study Suggests Politico

Researchers from the University of Oxford looked at over a million patient records and found that, two years after infection, patients who had recovered from COVID-19 were at higher risk of psychosis, dementia and “brain fog” compared to patients who recovered from other respiratory ailments. For some symptoms, there was an initial increase that leveled off. Anxiety and depression dropped to rates consistent with other respiratory illnesses after two months. But in the case of brain fog, for example, adults between the ages of 18 and 64 who had recovered from COVID-19 suffered from it at a 16% higher rate than patients with other respiratory illnesses. The difference was most pronounced in people aged over 65, where an increased risk for psychosis and dementia was also found. Evidence, mostly from patients in the US, suggests that minors are also affected. Children who recovered from COVID-19 were twice as likely to suffer from epilepsy or seizures and three times more likely to develop a psychotic disorder compared to those who recovered from the respiratory illness, although the absolute risk of the conditions remains low. The study, in The Lancet Psychiatry, showed that even the milder Omicron variant of the coronavirus that is currently prevalent carries similar long-term risks. Maxime Taquet, one of the study’s authors, noted that only patients who were sick enough to enter the health system and receive a COVID-19 diagnosis were included in the study, which underestimates those with only mild symptoms. However, the same is true for the comparison group of patients who recovered from other respiratory diseases. The study sought to “reveal what COVID, as a virus, does to you specifically, versus what other viruses that affect the same part of your body in a broadly similar way might do,” said lead author Paul Harrison. He added that the study was not designed to determine the biological mechanism by which the virus causes the increased risk of psychological and neurological disorder. The paper adds to the growing body of evidence showing the long-term damage caused by the coronavirus. The issue has concerned governments, which are spending money to research and treat the cluster of symptoms informally known as “long COVID,” a label that includes both neurological problems and fatigue and shortness of breath. The Institute for Health Metrics and Evaluation estimates that 3.7 percent of patients with COVID-19 develop a symptom after the disease, said Janet Diaz, WHO’s lead on the matter. Speaking at a conference on Wednesday, he said the average severity of conditions after COVID-19 is equivalent to that experienced by patients with severe neck pain, Crohn’s disease or the long-term effects of a traumatic brain injury. This article is part of POLITICO Pro The one-stop solution for policy professionals who combine the depth of POLITICO journalism with the power of technology Exclusively breaking scoops and ideas Customized policy information platform A high-level public affairs network

title: “Covid May Damage Brain Function Major Study Suggests Politico Klmat” ShowToc: true date: “2022-11-02” author: “Thomas Ryerson”

Researchers from the University of Oxford looked at over a million patient records and found that, two years after infection, patients who had recovered from COVID-19 were at higher risk of psychosis, dementia and “brain fog” compared to patients who recovered from other respiratory ailments. For some symptoms, there was an initial increase that leveled off. Anxiety and depression dropped to rates consistent with other respiratory illnesses after two months. But in the case of brain fog, for example, adults between the ages of 18 and 64 who had recovered from COVID-19 suffered from it at a 16% higher rate than patients with other respiratory illnesses. The difference was most pronounced in people aged over 65, where an increased risk for psychosis and dementia was also found. Evidence, mostly from patients in the US, suggests that minors are also affected. Children who recovered from COVID-19 were twice as likely to suffer from epilepsy or seizures and three times more likely to develop a psychotic disorder compared to those who recovered from the respiratory illness, although the absolute risk of the conditions remains low. The study, in The Lancet Psychiatry, showed that even the milder Omicron variant of the coronavirus that is currently prevalent carries similar long-term risks. Maxime Taquet, one of the study’s authors, noted that only patients who were sick enough to enter the health system and receive a COVID-19 diagnosis were included in the study, which underestimates those with only mild symptoms. However, the same is true for the comparison group of patients who recovered from other respiratory diseases. The study sought to “reveal what COVID, as a virus, does to you specifically, versus what other viruses that affect the same part of your body in a broadly similar way might do,” said lead author Paul Harrison. He added that the study was not designed to determine the biological mechanism by which the virus causes the increased risk of psychological and neurological disorder. The paper adds to the growing body of evidence showing the long-term damage caused by the coronavirus. The issue has concerned governments, which are spending money to research and treat the cluster of symptoms informally known as “long COVID,” a label that includes both neurological problems and fatigue and shortness of breath. The Institute for Health Metrics and Evaluation estimates that 3.7 percent of patients with COVID-19 develop a symptom after the disease, said Janet Diaz, WHO’s lead on the matter. Speaking at a conference on Wednesday, he said the average severity of conditions after COVID-19 is equivalent to that experienced by patients with severe neck pain, Crohn’s disease or the long-term effects of a traumatic brain injury. This article is part of POLITICO Pro The one-stop solution for policy professionals who combine the depth of POLITICO journalism with the power of technology Exclusively breaking scoops and ideas Customized policy information platform A high-level public affairs network

title: “Covid May Damage Brain Function Major Study Suggests Politico Klmat” ShowToc: true date: “2022-11-16” author: “Fannie Mensing”

Researchers from the University of Oxford looked at over a million patient records and found that, two years after infection, patients who had recovered from COVID-19 were at higher risk of psychosis, dementia and “brain fog” compared to patients who recovered from other respiratory ailments. For some symptoms, there was an initial increase that leveled off. Anxiety and depression dropped to rates consistent with other respiratory illnesses after two months. But in the case of brain fog, for example, adults between the ages of 18 and 64 who had recovered from COVID-19 suffered from it at a 16% higher rate than patients with other respiratory illnesses. The difference was most pronounced in people aged over 65, where an increased risk for psychosis and dementia was also found. Evidence, mostly from patients in the US, suggests that minors are also affected. Children who recovered from COVID-19 were twice as likely to suffer from epilepsy or seizures and three times more likely to develop a psychotic disorder compared to those who recovered from the respiratory illness, although the absolute risk of the conditions remains low. The study, in The Lancet Psychiatry, showed that even the milder Omicron variant of the coronavirus that is currently prevalent carries similar long-term risks. Maxime Taquet, one of the study’s authors, noted that only patients who were sick enough to enter the health system and receive a COVID-19 diagnosis were included in the study, which underestimates those with only mild symptoms. However, the same is true for the comparison group of patients who recovered from other respiratory diseases. The study sought to “reveal what COVID, as a virus, does to you specifically, versus what other viruses that affect the same part of your body in a broadly similar way might do,” said lead author Paul Harrison. He added that the study was not designed to determine the biological mechanism by which the virus causes the increased risk of psychological and neurological disorder. The paper adds to the growing body of evidence showing the long-term damage caused by the coronavirus. The issue has concerned governments, which are spending money to research and treat the cluster of symptoms informally known as “long COVID,” a label that includes both neurological problems and fatigue and shortness of breath. The Institute for Health Metrics and Evaluation estimates that 3.7 percent of patients with COVID-19 develop a symptom after the disease, said Janet Diaz, WHO’s lead on the matter. Speaking at a conference on Wednesday, he said the average severity of conditions after COVID-19 is equivalent to that experienced by patients with severe neck pain, Crohn’s disease or the long-term effects of a traumatic brain injury. This article is part of POLITICO Pro The one-stop solution for policy professionals who combine the depth of POLITICO journalism with the power of technology Exclusively breaking scoops and ideas Customized policy information platform A high-level public affairs network

title: “Covid May Damage Brain Function Major Study Suggests Politico Klmat” ShowToc: true date: “2022-12-12” author: “Dana Jimmerson”

Researchers from the University of Oxford looked at over a million patient records and found that, two years after infection, patients who had recovered from COVID-19 were at higher risk of psychosis, dementia and “brain fog” compared to patients who recovered from other respiratory ailments. For some symptoms, there was an initial increase that leveled off. Anxiety and depression dropped to rates consistent with other respiratory illnesses after two months. But in the case of brain fog, for example, adults between the ages of 18 and 64 who had recovered from COVID-19 suffered from it at a 16% higher rate than patients with other respiratory illnesses. The difference was most pronounced in people aged over 65, where an increased risk for psychosis and dementia was also found. Evidence, mostly from patients in the US, suggests that minors are also affected. Children who recovered from COVID-19 were twice as likely to suffer from epilepsy or seizures and three times more likely to develop a psychotic disorder compared to those who recovered from the respiratory illness, although the absolute risk of the conditions remains low. The study, in The Lancet Psychiatry, showed that even the milder Omicron variant of the coronavirus that is currently prevalent carries similar long-term risks. Maxime Taquet, one of the study’s authors, noted that only patients who were sick enough to enter the health system and receive a COVID-19 diagnosis were included in the study, which underestimates those with only mild symptoms. However, the same is true for the comparison group of patients who recovered from other respiratory diseases. The study sought to “reveal what COVID, as a virus, does to you specifically, versus what other viruses that affect the same part of your body in a broadly similar way might do,” said lead author Paul Harrison. He added that the study was not designed to determine the biological mechanism by which the virus causes the increased risk of psychological and neurological disorder. The paper adds to the growing body of evidence showing the long-term damage caused by the coronavirus. The issue has concerned governments, which are spending money to research and treat the cluster of symptoms informally known as “long COVID,” a label that includes both neurological problems and fatigue and shortness of breath. The Institute for Health Metrics and Evaluation estimates that 3.7 percent of patients with COVID-19 develop a symptom after the disease, said Janet Diaz, WHO’s lead on the matter. Speaking at a conference on Wednesday, he said the average severity of conditions after COVID-19 is equivalent to that experienced by patients with severe neck pain, Crohn’s disease or the long-term effects of a traumatic brain injury. This article is part of POLITICO Pro The one-stop solution for policy professionals who combine the depth of POLITICO journalism with the power of technology Exclusively breaking scoops and ideas Customized policy information platform A high-level public affairs network

title: “Covid May Damage Brain Function Major Study Suggests Politico Klmat” ShowToc: true date: “2022-12-06” author: “James Rodriguez”

Researchers from the University of Oxford looked at over a million patient records and found that, two years after infection, patients who had recovered from COVID-19 were at higher risk of psychosis, dementia and “brain fog” compared to patients who recovered from other respiratory ailments. For some symptoms, there was an initial increase that leveled off. Anxiety and depression dropped to rates consistent with other respiratory illnesses after two months. But in the case of brain fog, for example, adults between the ages of 18 and 64 who had recovered from COVID-19 suffered from it at a 16% higher rate than patients with other respiratory illnesses. The difference was most pronounced in people aged over 65, where an increased risk for psychosis and dementia was also found. Evidence, mostly from patients in the US, suggests that minors are also affected. Children who recovered from COVID-19 were twice as likely to suffer from epilepsy or seizures and three times more likely to develop a psychotic disorder compared to those who recovered from the respiratory illness, although the absolute risk of the conditions remains low. The study, in The Lancet Psychiatry, showed that even the milder Omicron variant of the coronavirus that is currently prevalent carries similar long-term risks. Maxime Taquet, one of the study’s authors, noted that only patients who were sick enough to enter the health system and receive a COVID-19 diagnosis were included in the study, which underestimates those with only mild symptoms. However, the same is true for the comparison group of patients who recovered from other respiratory diseases. The study sought to “reveal what COVID, as a virus, does to you specifically, versus what other viruses that affect the same part of your body in a broadly similar way might do,” said lead author Paul Harrison. He added that the study was not designed to determine the biological mechanism by which the virus causes the increased risk of psychological and neurological disorder. The paper adds to the growing body of evidence showing the long-term damage caused by the coronavirus. The issue has concerned governments, which are spending money to research and treat the cluster of symptoms informally known as “long COVID,” a label that includes both neurological problems and fatigue and shortness of breath. The Institute for Health Metrics and Evaluation estimates that 3.7 percent of patients with COVID-19 develop a symptom after the disease, said Janet Diaz, WHO’s lead on the matter. Speaking at a conference on Wednesday, he said the average severity of conditions after COVID-19 is equivalent to that experienced by patients with severe neck pain, Crohn’s disease or the long-term effects of a traumatic brain injury. This article is part of POLITICO Pro The one-stop solution for policy professionals who combine the depth of POLITICO journalism with the power of technology Exclusively breaking scoops and ideas Customized policy information platform A high-level public affairs network